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Enlarged Breast Tissue in Men

👤by MedicineNet.com 0 comments 🕔Monday, December 1st, 2014

The appearance of enlarged breast tissue in men can be either due to the true enlargement of actual breast tissue or a result of deposition of fat in the pectoral area of the chest. True enlargement of the gland tissue of the breast is due to hormonal fluctuations or imbalances. Breast enlargement in men due to this condition is referred to as gynecomastia. However, especially in obese or overweight men, there can be an increase in fatty tissue over the breast area. This condition, which is not the same as true enlargement of male breast tissue, creates an appearance of enlarged breasts and is known as pseudogynecomastia.

Doctors can usually tell whether or not enlarged breasts in men are due to true gynecomastia by the physical examination, but sometimes other testing is required. True gynecomastia (enlargement of breast tissue), in men is not uncommon and can be seen in normal, healthy boys during puberty or in men as they age. Medical conditions that interfere with the body's hormonal balance can also cause breast enlargement in men.

The greatest risk factor for the development of fatty tissue in the breast area is obesity and weight gain. Obesity is defined by the body mass index; adults with a BMI between 25 and 30 are considered overweight; those with a BMI >30 are considered to be obese. Obesity is a major public health problem in the U.S. and accounts for significant morbidity (disease states with reduced quality of life) and mortality (deaths).

Medically Reviewed by a Doctor on 12/1/2014

Medical Author:

Melissa Conrad Stöppler, MD

Melissa Conrad Stöppler, MD

Melissa Conrad Stöppler, MD, is a U.S. board-certified Anatomic Pathologist with subspecialty training in the fields of Experimental and Molecular Pathology. Dr. Stöppler's educational background includes a BA with Highest Distinction from the University of Virginia and an MD from the University of North Carolina. She completed residency training in Anatomic Pathology at Georgetown University followed by subspecialty fellowship training in molecular diagnostics and experimental pathology.

Medical Editor:

William C. Shiel Jr., MD, FACP, FACR

William C. Shiel Jr., MD, FACP, FACR

Dr. Shiel received a Bachelor of Science degree with honors from the University of Notre Dame. There he was involved in research in radiation biology and received the Huisking Scholarship. After graduating from St. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. He is board-certified in Internal Medicine and Rheumatology.

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